Synonyms for anosognosia or Related words with anosognosia

hemihypacusis              hemispatial              palinopsia              anomic              prosopagnosia              derealization              hemianopia              hemihypesthesia              hemiparesis              aphasias              dysgnosia              hemiballismus              agnosia              agnosias              hyperarousal              parosmia              acalculia              aboulia              agraphia              apperceptive              dysarthria              anosmia              paresthesias              misophonia              dyscalculia              capgras              obtundation              hemianopsia              dysmetria              micropsia              paraphrenia              moodiness              aphasic              dysphasia              combativeness              malingering              ideomotor              sociopathy              quadriplegia              hyporeflexia              amusia              tetraplegia              presyncope              amnesias              paraphasia              scotomata              coprolalia              hypoesthesia              hypochondria              confabulation             



Examples of "anosognosia"
When assessing the causes of anosognosia within stroke patients, CT scans have been used to assess where the greatest amount of damage is found within the various areas of the brain. Stroke patients with mild and severe levels of anosognosia (determined by response to an anosognosia questionnaire) have been linked to lesions within the temporoparietal and thalamic regions, when compared to those who experience moderate anosognosia, or none at all. In contrast, after suffering a stroke, people who have moderate anosognosia have a higher frequency of lesions involving the basal ganglia, compared to those with mild or severe anosognosia.
Anosognosia is a lack of awareness or understanding of the loss of function caused by the brain injury and is common in individuals who have suffered a right hemisphere stroke. Because patients with anosognosia may be unaware of their deficits, they may be less likely to seek treatment once they are released from the hospital. The lack of proper treatment could lead to higher levels of dependency later on. In order to make functional recovery gains, right hemisphere stroke survivors should receive rehabilitation services, so patients with anosognosia should be encouraged to seek out additional treatment. However, due to the anosognosia, these patients often report a higher perceived quality of life than other right hemisphere stroke survivors because of the unawareness of the resulting deficits (Daia et al., 2014). Patients with smaller lesions often recover faster from anosognosia than patients with larger lesions resulting in anosognosia (Hier et al., 1983).
There are also studies showing that the maneuver of vestibular stimulation could temporarily improve both the syndrome of spatial unilateral neglect and of anosognosia for left hemiplegia. Combining the findings of hemispheric asymmetry to the right, association with spatial unilateral neglect, and the temporal improvement on both syndromes, it is suggested there can be a spatial component underlying the mechanism of anosognosia for motor weakness and that neural processes could be modulated similarly. There were some cases of anosognosia for right hemiplegia after left hemisphere damage, but the frequency of this type of anosognosia has not been estimated.
Hemiasomatognosia is a subtype of Anosognosia in which the person suffering from hemiplegia neglects one half of his body.
Anosognosia is a condition in which a person who suffers certain disability seems unaware of the existence of his or her disability. Hemiasomatognosia is a subtype of anosognosia in which the person suffering from hemiplegia neglects one half of his body.
Somatoparaphrenia has been reported to occur predominately in the left arm of one's body, and it is often accompanied by left-sided paralysis and anosognosia (denial or lack of awareness) of the paralysis. The link between somatoparaphrenia and paralysis has been documented in many clinical cases and the question arises as to whether paralysis, anosognosia or both are necessary for somatoparaphrenia to occur.
Marcel, A.J., Tegnr, R & Nimmo-Smith, I., (2004). Anosognosia for plegia: specificity, extension, partiality, and disunity of bodily unawareness. Cortex, 40, 19–40.
Although largely used to describe unawareness of impairment after brain injury or stroke, the term 'anosognosia' is occasionally used to describe the lack of insight shown by some people who suffer from anorexia nervosa. They do not seem to recognize that they suffer from a mental illness. There is evidence that 'anosognosia' related to schizophrenia may be the result of frontal lobe damage. E. Fuller Torrey, a psychiatrist and schizophrenia researcher, has stated that among those with schizophrenia and bipolar disorder, anosognosia is the most prevalent reason for not taking medications.
A similar situation can happen on patients with anosognosia for cognitive deficits after traumatic brain injury when monitoring their errors during the tasks regarding their memory and attention (online emergent awareness) and when predicting their performance right before the same tasks (online anticipatory awareness). It can also occur among patients with dementia and anosognosia for memory deficit when prompted with dementia-related words, showing possible pre-attentive processing and implicit knowledge of their memory problems. More interestingly, patients with anosognosia may overestimate their performance when asked in first-person formed questions but not from a third-person perspective when the questions referring to others.
Anosognosia is relatively common following different causes of brain injury, such as stroke and traumatic brain injury; for example, anosognosia for hemiparesis, (weakness of one side of the body) with onset of acute stroke is estimated at between 10% and 18%. However, it can appear to occur in conjunction with virtually any neurological impairment. It is more frequent in the acute than in the chronic phase and more prominent for assessment in the cases with right hemispheric lesions than with the left. Anosognosia is not related to global mental confusion, cognitive flexibility, other major intellectual disturbances, or mere sensory/perceptual deficits.
2. May be related to an affective communication disorder and defective arousal. These emotional disorders cannot account for the verbal explicit denial of illness of anosognosia.
Thus we have the twin symptoms of anosognosia (or lack of awareness of defect) and confabulation, the latter affecting both speech and behaviour.
Nimmo-Smith, I, Marcel, A.J., & Tegnr, R. (2005). A diagnostic test of unawareness of bilateral motor abilities in anosognosia for hemiplegia. Journal of Neurology, Neurosurgery and Psychiatry, 76, 1167–1169.
Anosognosia can be selective in that an affected person with multiple impairments may seem unaware of only one handicap, while appearing to be fully aware of any others. This is consistent with the idea that the source of the problem relates to spatial representation of the body. For example, anosognosia for hemiplegia, or the paralysis of one side of the body, may occur with or without intact awareness of visuo-spatial unilateral neglect. This phenomenon of double dissociation can be an indicator of domain-specific disorders of awareness modules, meaning that in anosognosia, brain damage can selectively impact the self-monitoring process of one specific physical or cognitive function rather than a spatial location of the body.
Clinically, anosognosia is often assessed by giving patients an anosognosia questionnaire in order to assess their metacognitive knowledge of deficits. However, neither of the existing questionnaires applied in the clinics are designed thoroughly for evaluating the multidimensional nature of this clinical phenomenon; nor are the responses obtained via offline questionnaire capable of revealing the discrepancy of awareness observed from their online task performance. The discrepancy is noticed when patients showed no awareness of their deficits from the offline responses to the questionnaire but demonstrated reluctance or verbal circumlocution when asked to perform an online task. For example, patients with anosognosia for hemiplegia may find excuses not to perform a bimanual task even though they do not admit it is because of their paralyzed arms.
Anosognosia (, ; from Ancient Greek ἀ- "a-", "without", νόσος "nosos", "disease" and γνῶσις "gnōsis", "knowledge") is a deficit of self-awareness, a condition in which a person who suffers some disability seems unaware of the existence of their disability. It was first named by the neurologist Joseph Babinski in 1914. Anosognosia results from physiological damage to brain structures, typically to the parietal lobe or a diffuse lesion on the fronto-temporal-parietal area in the right hemisphere, and is thus a neurological disorder. While this distinguishes the condition from denial, which is a psychological defense mechanism, attempts have been made at a unified explanation. Anosognosia is sometimes accompanied by asomatognosia, a form of neglect in which patients deny ownership of their limbs.
Weinstein: Weinstein’s viewpoint of jargon illustrates just the basic rambling and incoherent but structurally intact speech. It does not include details about neologisms and paraphasias. He and his coworkers view jargon as a positive symptom of aphasia, and as an adaptive behavior and form of denial, or anosognosia, in the presence of language deficit.
Anosognosia is also closely related to other cognitive dysfunctions that may impair the capacity of an individual to continuously participate in treatment. Other research has suggested that attitudes toward treatment can improve after involuntary treatment and that previously committed patients tend later to seek voluntary treatment.
William Hirstein received his Ph.D. in philosophy at the University of California, Davis under the direction of Richard Wollheim. He then did post-doctoral work under the supervision of Patricia Churchland and Vilayanur S. Ramachandran at the University of California, San Diego, exploring neurological syndromes that lead to confabulation, such as in split-brain patients, and patients with anosognosia or Capgras delusion.
Marcel's later research focuses on consciousness and phenomenal experience. His research on neglect and anosognosia for hemiplegia has continued his work on lack of conscious awareness in neurological patients, investigating similarities between a subgroup of delusional anosognosic patients and psychotic delusional patients.