Synonyms for hypersalivation or Related words with hypersalivation
Examples of "hypersalivation"
, a prominent autonomic manifestation, is often associated with hemifacial seizures, oro-pharyngo-laryngeal symptoms and speech arrest.
is not just frothing:
also often precedes emesis (vomiting), where it accompanies nausea (a feeling of needing to vomit).
Hemifacial seizures are often associated with an inability to speak and
is optimally treated by treating or avoiding the underlying cause. Mouthwash and tooth brushing may have drying effects.
According to the ASPCA, yarrow is toxic to dogs, cats, and horses, causing vomiting, diarrhea, depression, anorexia, and
can contribute to drooling if there is an inability to keep the mouth closed or in difficulty in swallowing the excess saliva (dysphagia).
Signs of respiratory disease include tachycardia and tachypnea with pyrexia, dyspnea, mucoid nasal discharge,
and abnormal lung sounds. Systemic signs such as lethargy and anorexia are seen.
(also called ptyalism or sialorrhea) is excessive production of saliva. It has also been defined as increased amount of saliva in the mouth, which may also be caused by decreased clearance of saliva.
Symptoms can be aggravated by hunger, fatigue, prenatal vitamins (especially those containing iron), and diet. Many people with HG are extremely sensitive to odors in their environment; certain smells may exacerbate symptoms. This is known as hyperolfaction. Ptyalism, or
, is another symptom experienced by some women suffering from HG.
The first phase of the poisoning is characterized by facial and extremity paresthesia, the victim feels tingling and/or numbness on the face, tongue, lips and other body extremities. The victim may also have over sweating, headache, dizziness, speech problems,
, vomiting, nausea, diarrhea, abdominal pain, movement disorders and a feeling of weakness, cyanosis to extremities and lips, petechial hemorrhages on the body.
Other seizure types: Despite prominent
, focal seizures with primarily autonomic manifestations (autonomic seizures) are not considered part of the core clinical syndrome of Rolandic epilepsy. However, some children may present with independent autonomic seizures or seizures with mixed Rolandic-autonomic manifestations including emesis as in Panayiotopoulos syndrome.
There are the muscarinic signs, which are effects that are similar to stimulation on postganglionic parasympathic nerves. These signs are usually the first to manifest. They consist of
, the secretion of tears, sweating, and nasal discharge. Dilation of the pupil (miosis), impaired breathing (dyspnea), vomiting, diarrhea, and frequency of urination are also signs in that may occur in this category.
While clozapine is a muscarinic antagonist at the M1, M2, M3, and M5 receptors, clozapine is a full agonist at the M4 subset. Because M4 is highly expressed in the salivary gland, its M4 agonist activity is thought to be responsible for the
The venom produces mainly cardiopulmonary abnormalities like circulatory derangements, myocarditis and changes in cardiac sarcolemmal ATPase and by these abnormalities it can finally cause death. In rural India the scorpion and its venom is a commonly known factor of children's death. The venom initially causes transient cholinergic stimulation (vomiting, profuse sweating, bradycardia, priapism,
, and hypotension) which is followed by sustained adrenergic hyperactivity (hypertension, tachycardia, and myocardial failure). The adrenergic phase but not the cholinergic phase is a dose-dependent phenomenon.
For cold-blooded animals the effects are slightly different. In a study with frogs, acute exposure caused a depression in the amount of erythocytes in the blood. There was also a reduction of white bloodcells, especially the neutrophil granulocytes and lymphocytes. There was no visible damage to the bloodvessels to explain the loss of blood cells. Further there were no signs like
or tears like in warm-blooded animals. Though there was hypotonia leading to paralysis.
The median lethal dose of oral exposure in rat is 85 mg/kg and that of dermal is 390 mg/kg.。It is a weak acetylcholinesterase inhibitor. It is taken by not only oral and inhalation but skin and it causes toxic symptoms peculiar to organophosphorus compounds such as miosis,
, hyperhidrosis, chest pressure, pulmonary edema and fecal incontinence. It is flammable and decomposes to toxic gases such as phosphorus oxides, sulfur oxides and nitrogen oxides. It is harmful especially to water creatures.
Other symptoms may include
when attacks are localized in the mandibular region, or leg weakness after foot trauma. A prominent non-physical symptom are tonic non-epileptic seizures. Such seizures are more common in infancy and childhood than during adulthood. In older children, inconsolable screaming usually precedes such attack, followed by apnea, paleness, and stiffness. Such stiffness can last from seconds to a few minutes.
destructiveness, faecal or urinary elimination,
or vocalisation. Dogs from single-owner homes are approximately 2.5 times more likely to have separation anxiety compared to dogs from multiple-owner homes. Furthermore, sexually intact dogs are only one third as likely to have separation anxiety as neutered dogs. The sex of dogs and whether there is another pet in the home do not have an effect on separation anxiety.
ANTU causes local gastric irritation in animals and when it is absorbed, it increases permeability of the lung capillaries in all animals. The symptoms that the animals present after absorption of alpha-naphthyl thiourea are first weakness, ataxia, weak pulse and subnormal temperatures. Afterwards, they have the following symptoms: vomiting,
, coughing and severe pulmonary edema. In the most cases a pale, mottled liver and damaged kidneys are found in animals which have ingested ANTU.
The toxins of the cigarette butt can cause health problems in animals like vomiting, tremors and
. Veterinary Medicine published a case of a 10-year-old female Labrador retriever ingesting cigarette butts. The Labrador vomited several times and had increased blood urea nitrogen, total protein, and albumin concentrations with hemolysis and lipemia also observed. Apomorphine hydrochloride and activated charcoal had to be administered along with other fluids. 5 days after the incident, the dog’s health returned to normal. The toxic level of nicotine in a dog or cat is reported as 20–100 mg which is about one to five cigarettes.
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