Synonyms for hypothyroid or Related words with hypothyroid
Examples of "hypothyroid"
During the hyperthyroid phase, iodine uptake is suppressed, while during the
phase, uptake is increased.
The initial phase of hyperthyroid symptoms occurs transiently about two to six months postpartum. Typical symptoms include irritability, nervousness, palpitations, and heat intolerance. Hormonal disturbances during this phase tend to occur with lower intensity compared with the
phase. As a result, the hyperthyroid phase may pass undetected. The second phase of
symptoms is also transient and can occur anytime within the three- to twelve-month period postpartum. Women in this phase experience low energy, poor memory, impaired concentration, carelessness, dry skin, cold intolerance, and general aches and pains. After one year postpartum, euthyroid function resumes. Any case with
symptoms extending beyond one year postpartum is not considered postpartum thyroiditis.
Also known as Debre–Semelaigne syndrome or cretinism-muscular hypertrophy,
myopathy, hypothyroidism-large muscle syndrome, hypothyreotic muscular hypertrophy in children, infantile myxoedema-muscular hypertrophy, myopathy-myxoedema syndrome, myxoedema-muscular hypertrophy syndrome, myxoedema-myotonic dystrophy syndrome.
Riedel's thyroiditis is classified as rare. Most patients remain euthyroid, but approximately 30% of patients become
and very few patients are hyperthyroid. It is most seen in women.
Note: certain symptoms and physical changes can be seen in both
and hyperthyroid disorders—fatigue, fine / thinning hair, menstrual cycle irregularities, muscle weakness / aches (myalgia), and different forms of myxedema.
There is increased sensitivity to radioiodine therapy in thyroids appearing on ultrasound scans as more uniform (hypoechogenic), due to densely packed large cells, with 81% later becoming
, compared to just 37% in those with more normal scan appearances (normoechogenic).
After women give birth, about 5% develop postpartum thyroiditis which can occur up to nine months afterwards. This is characterized by a short period of hyperthyroidism followed by a period of hypothyroidism; 20–40% remain permanently
Recent research suggested the existence of an additional feedforward motif linking TSH release to deiodinase activity in humans. The existence of this TSH-T3 shunt could explain why deiodinase activity is higher in
patients and why a minor fraction of affected individuals may benefit from substitution therapy with T3.
Per the U.S. FDA, liothyronine is categorized as Pregnancy Category A. Thyroid hormone is minimally transferred to the fetus or placenta, however as of October 2014, studies have not shown any adverse effects to the fetus.
mothers should continue to take thyroid hormone replacement therapy throughout pregnancy to avoid adverse events.
During his years of practice, Barnes also began to believe that virtually all his
patients had concomitant undiagnosed adrenal insufficiency. Based on this speculation, he routinely prescribed an accompanying dose of the synthetic corticosteroid prednisone. Barnes argued that it was mandatory to give prednisone to patients with a systolic blood pressure below 100.
In pregnancy, subclinical hypothyroidism is defined as a TSH between 2.5 and 10 mIU/l with a normal thyroxine level, while those with TSH above 10 mIU/l are considered to be overtly
even if the thyroxine level is normal. Antibodies against TPO may be important in making decisions about treatment, and should, therefore, be determined in women with abnormal thyroid function tests.
Thiocyanate is a potent competitive inhibitor of the thyroid sodium-iodide symporter. Iodine is an essential component of thyroxine. Since thiocyanates will decrease iodide transport into the thyroid follicular cell, they will decrease the amount of thyroxine produced by the thyroid gland. As such, foodstuffs containing thiocyanate are best avoided by
She met her husband, 58 lb Peter Robinson, a fellow sideshow performer known and billed as "the human skeleton", while filming the cult classic "Freaks". They had two children. Smith was diagnosed as
in 1951 and died shortly after of a heart attack.
One study showed infants born to treated
mothers had abnormal thyroid function compared to matched controls. Therefore, it is advised to monitor T4 levels throughout the pregnancy in case treatment dosages should be increased to accommodate both the mother’s and fetus’s thyroid hormone requirements. If the supply of T4 is insufficient the mother may be at risk for preeclampsia and preterm delivery.
The tongue may show a diffuse, smooth generalized enlargement. The face may show maxillary hypoplasia causing relative mandibular prognathism. Apparent macroglossia can also occur in Down syndrome. The tongue has a papillary, fissured surface. Macroglossia may be a sign of
GD correlates with body mass index and thyrotropin levels in humans, and it is reduced in nonthyroidal illness with hypodeiodination. Recent research revealed total deiodinase activity to be higher in
patients, which may ensue from the existence of an effective TSH-deiodinase axis or TSH-T3 shunt.
The hypersensitivity syndrome is characterized by a skin eruption that is initially morbilliform. The rash may also be a severe Stevens-Johnson syndrome or toxic epidermal necrolysis. Systemic manifestations occur at the time of skin manifestations and include eosinophilia, hepatitis, and interstitial nephritis. However, a subgroup of patients may become
as part of an autoimmune thyroiditis up to 2 months after the initiation of symptoms.
In 1971, the thyroid stimulating hormone (TSH) radioimmunoassay was developed, which was the most specific marker for assessing thyroid status in patients. Many people who were being treated based on basal metabolic rate, minimizing
symptoms, or based on serum protein-bound iodine, were found to have excessive thyroid hormone. The following year, in 1972, a T3 radioimmunoassay was developed, and in 1974, a T4 radioimmunoassay was developed.
patients on thyroxine, measurement of TSH alone is generally considered sufficient. An increase in TSH above the normal range indicates under-replacement or poor compliance with therapy. A significant reduction in TSH suggests over-treatment. In both cases, a change in dose may be required. A low or low-normal TSH value may also signal pituitary disease. TSH measurements could not be applied any more, however, treatment would have to be continued.
In cardiomyopathy the heart reverts to a fetal gene programming due to the overload of the heart. Like during fetal development, thyroid hormone levels are low in the overloaded heart tissue in a local
state, with low levels of deiodinase 1 and deiodinase 2. Although deiodinase 3 levels in a normal heart are generally low, in cardiomyopathy deiodinase 3 activity is increased to decrease energy turnover and oxygen consumption.
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