Synonyms for nephrolithiasis or Related words with nephrolithiasis

urolithiasis              hypercalciuria              ureterolithiasis              calculous              lithiasis              oxalosis              osteodystrophy              hypereninemic              cushings              nephrocalcinosis              tophaceous              choledocholithiasis              hyperuricemia              cystolithiasis              gallstones              calciphylaxis              hydronephrosis              hyponatremia              gonadotropinoma              hypercalcaemia              tubulointestitial              hyperuricosuria              crystalluria              hypocalciuria              hyperphosphatemia              cholelithiasis              sialolithiasis              gallstone              rachitis              hypouricemia              prostatomegaly              hematuria              hypernatremia              adenomyomas              hypocalcemia              hyperthyroidism              cystinuria              lipomembranous              uremia              hypoadrenocorticism              addisons              plumbism              cholescystitis              proteinuric              hepatorenal              cholecystolithiasis              cholestatic              serositis              menieres              cholestasia             



Examples of "nephrolithiasis"
In a 2009 study of 683 children diagnosed in Beijing in 2008 with nephrolithiasis and 6,498 children without nephrolithiasis aged < 3 years, investigators found that in children exposed to melamine levels < 0.2 mg/kg per day, the risk for nephrolithiasis was 1.7 times higher than in those without melamine exposure, suggesting that the risk of melamine-induced nephrolithiasis in young children starts at a lower intake level than the levels recommended by the World Health Organization.
Chronic hyperuricosuria is associated with gout and uric acid nephrolithiasis. However, both conditions can occur in the absence of hyperuricosuria. Treatment of gout with uricosuric drugs can lead to uric acid nephrolithiasis.
Mutations in this gene are associated with hypophosphatemia nephrolithiasis/osteoporosis 1.
It can result in nephrolithiasis (kidney stones), acute renal failure and permanent kidney damage.
Renal papillary damage has also been associated with nephrolithiasis and can be quantified according to the papillary grading score, which accounts for contour, pitting, plugging and randall plaque.
Sevelamer can significantly reduce serum uric acid. This reduction has no known detrimental effect and several beneficial effects, including reducing hyperuricemia, uric acid nephrolithiasis, and gout.
The reduced enzyme function can be caused by a rare inherited autosomal recessive disorder known as primary hyperoxaluria type II (PH2). This condition can cause nephrolithiasis (kidney stone), nephrocalcinosis and renal failure.
The surgery is indicated for all patients that are diagnosed with hyperparathyroidism with or without symptoms, especially in younger patients. In some cases the surgery works as therapy for nephrolithiasis, bone changes, and neuromuscular symptoms.
Acute hyperuricosuria is a common complication of tumor lysis syndrome. This syndrome appears not to contribute to gout and to uric acid nephrolithiasis, which is evidence that these two conditions have chronic, not acute, causes.
Hyperglycinuria is another disorder affecting reabsorption of glycine and imino acids, similar to iminoglycinuria and considered to be a heterozygous form. When accompanied by a specific type of kidney stone (nephrolithiasis), it is sometimes referred to as "iminoglycinuria, type II".
"Renal calculus" is from the Latin "rēnēs" meaning "kidneys," and "calculus" meaning "pebble". Lithiasis (stone formation) in the kidneys is called nephrolithiasis (), from "nephro-" meaning kidney + "-lith" meaning stone and "-iasis" meaning disorder.
STEFAN - The spring known since 1822. The bi-carbonate-chloride-sodium-iodide acidic water. Recommended, among others, in urinary tract inflammations, nephrolithiasis, catarrhs of nose, throat, bronchi inflammations, asthma, emphysema.
Dent disease 2 (nephrolithiasis type 2) is associated with the "OCRL" gene. Both Lowe syndrome (oculocerebrorenal syndrome) and Dent disease can be caused by truncating or missense mutations in "OCRL".
There are a number of documented cases of historical figures and distinguished members of society who were kidney stone formers. This condition is caused by nephrolithiasis, which are more commonly known as kidney stones, or urolithiasis, where the stone forms in the urinary system. These are crystal deposits that can accrete in the urinary system when certain chemical substances become concentrated in the urine. Among the symptoms associated with nephrolithiasis are intense colicky pain, nausea, fever, chills, and the reduction or blockage of urine flow. Historically, the condition of having a kidney or bladder stone was referred to as "the stone" or "the gravel".
A large number of mutations of HPRT are known. Mutations that only mildly decrease the enzyme's function do not normally cause the severe form of LNS, but do produce a milder form of the disease which still features purine overproduction accompanied by susceptibility to gout and uric acid nephrolithiasis.
Acute uric acid nephropathy (AUAN) due to hyperuricosuria has been a dominant cause of acute kidney failure but with the advent of effective treatments for hyperuricosuria, AUAN has become a less common cause than hyperphosphatemia. Two common conditions related to excess uric acid, gout and uric acid nephrolithiasis, are not features of tumor lysis syndrome.
In kidney stones, calcium oxalate is the most common mineral type (see Nephrolithiasis). Uric acid is the second most common mineral type, but an "in vitro" study showed uric acid stones and crystals can promote the formation of calcium oxalate stones.
If neither hyperuricemia nor gout is present, then the risk of uric acid nephrolithiasis can be reduced by the use of antiuricosuric drugs. One should also consider eating a low purine diet. Additionally, making the urine pH more alkaline is protective.
Idiopathic hypouricemia usually requires no treatment. In some cases, hypouricemia is a medical sign of an underlying condition that does require treatment. For example, if hypouricemia reflects high excretion of uric acid into the urine (hyperuricosuria) with its risk of uric acid nephrolithiasis, the hyperuricosuria may require treatment.
Often, aggressive treatment is unnecessary for people with MSK disease that does not cause any symptoms (asymptomatic). In such cases, treatment may consist of maintaining adequate fluid intake, with the goal of decreasing the risk of developing kidney stones (nephrolithiasis). Cases of recurrent kidney stone formation may warrant evaluation for possible underlying metabolic abnormalities.