Synonyms for poikiloderma or Related words with poikiloderma

civatte              keratoderma              chilblains              atrophicans              mucinosis              follicularis              vasculare              keratopathy              planopilaris              vegetans              telangiectasias              ecchymoses              keloidalis              pretibial              mucinosa              livedoid              parapsoriasis              ichtyosis              maculopapular              fulminans              fibroepithelioma              congenitale              chorioretinitis              melasma              cellulites              leucoderma              hypomelanotic              faciale              erythroderma              telangiectatic              chloasma              episcleritis              chalazion              xerophthalmia              trichodiscomas              centrifugum              xanthomata              decalvans              trichiasis              pustulosis              proptosis              exophthalmos              petechiae              pterygium              leukoderma              folliculitis              sclevitis              papulosquamous              neurodermatitis              xerotic             



Examples of "poikiloderma"
Kindler syndrome (also known as "Bullous acrokeratotic poikiloderma of Kindler and Weary,", "Congenital poikiloderma with blisters and keratoses," "Congenital poikiloderma with bullae and progressive cutaneous atrophy," "Hereditary acrokeratotic poikiloderma," "Hyperkeratosis–hyperpigmentation syndrome," "Acrokeratotic poikiloderma," and "Weary–Kindler syndrome") is a rare congenital disease of the skin caused by a mutation in the KIND1 gene.
Hereditary sclerosing poikiloderma is an autosomal dominant conditions with skin changes consisting of generalized poikiloderma appearing in childhood.
Albeit difficult, treatment of poikiloderma involves the delivery of multiple wavelengths of intense pulsed light (IPL) to the affected area.
Poikiloderma is a skin condition that consists of areas of hypopigmentation, hyperpigmentation, telangiectasias and atrophy.
The skin is normal at birth. Between 3 and 6 months of age, the affected carrier develops poikiloderma on the cheeks. This characteristic “rash” that all RTS carriers have can develop on the arms, legs and buttocks. “Poikiloderma consists of areas of increased and decreased pigmentation, prominent blood vessels, and thinning of the skin”
Rothmund–Thomson syndrome (RTS), also known as poikiloderma atrophicans with cataract or poikiloderma congenitale, is a rare autosomal recessive skin condition originally described by August von Rothmund (1830–1906) in 1868. Matthew Sydney Thomson (1894–1969) published further descriptions in 1936.
Rothmund-Thomson syndrome, also known as poikiloderma congenitale, is characterized by premature aging, skin and skeletal abnormalities, rash, poikiloderma, juvenile cataracts, and a predisposition to cancers such as osteosarcomas. Chromosomal rearrangements causing genomic instability are found in the cells of Rothmund-Thomson syndrome patients.
Poikiloderma vasculare atrophicans, or PVA, indicates that extra or altered skin pigmentation ("poikiloderma") is occurring, associated with heightened visibity of capillaries ("vasculare", referring to telangiectasia) under the skin, related to thinning and wasting away ("atrophicans") of the skin and its tissue. Telangiectasia is an enlargement of capillaries underneath the skin.
In modern consideration and usage, the solitary term "poikiloderma" has also come to represent all three elements of PVA. When skin diseases and disorders or skin conditions described as dermatoses contain the term poikiloderma in their assessment or diagnosis (such as with Bloom syndrome), this can sometimes be an erroneous usage of the term. Discretion has been advised. Usage of the entire term "poikiloderma vasculare atrophicans" may also be reserved to indicate it as the primary condition affecting the skin in cases where the disorder associated with it is secondary.
The exact cause of poikiloderma is unknown; however, extended sun exposure, namely the ultraviolet light emitted by the sun, is the primary factor.
Poikiloderma and Dermatoheliosis are treated by desquamation (skin peeling). There is no single effective treatment for HS. The recommended treatments include antibiotics, antiandrogens, corticosteroids, ciclosporins, and TNF inhibitors.
Poikiloderma is most frequently seen on the chest or the neck, characterized by red colored pigment on the skin that is commonly associated with sun damage.
Some of the other clinical characteristics sometimes associated with this disorder are growth retardation and poikiloderma. Although the presentation of BGS may differ between individuals, these characteristics are often observed. People with BGS may have stunted growth, short stature and misshapen kneecaps. Poikiloderma may also be present in people with this syndrome, meaning that their skin may have regions of hyperpigmentation and hypopigmentation, or regions where the skin is missing (atrophy).
Poikiloderma of Civatte is a cutaneous condition and refers to reticulated red to red-brown patches with telangiectasias. It is identifiable as the reddish brown discoloration on sides of the neck, usually on both sides. It is more common with women than men and more commonly effects middle-aged to elderly women. "Poikiloderma" is basically a change of the skin due to dilation of the blood vessels in the neck. "Civatte" was the French dermatologist who first identified it in the 1920s.
IPL is employed in the treatment of a range of dermatological conditions including photodamage induced dyspigmentation and vascular changes, poikiloderma of Civatte, rosacea, acne vulgaris, sebaceous gland hyperplasia, broken capillaries/telangiectases, vascular lesions (small blood vessels), pigmented lesions (freckles, liver spots, birth marks ), melasma, actinic keratosis, photorejuvenation, basal cell carcinoma, and Bowen's disease (squamous cell carcinoma).
Prurigo nodularis. "Prurigo nodularis" has been identified with coeliac disease. Rothmund-Thomson syndrome. Rothmund-Thomson syndrome, or poikiloderma congenitale, is a rare disorder, generally attributed to mutations of the RECQL4 helicase gene on 8q24 with features that include photosensitivity and poikilodermatous skin changes, etc., and has been reported in one celiac patient.
Poikiloderma of Civatte, a condition of dilated blood vessels and red to red-brown spots, is common to the upper part of the cleavage, especially for those who wear a sports bra or push-up bra for prolonged periods, and commonly affects fair skinned middle-aged to elderly women.
Poikiloderma vasculare atrophicans (PVA), sometimes referred to as parapsoriasis variegata or parapsoriasis lichenoides is a cutaneous condition (skin disease) characterized by hypo- or hyperpigmentation (diminished or heightened skin pigmentation, respectively), telangiectasia and skin atrophy. Other names for the condition include prereticulotic poikiloderma and atrophic parapsoriasis. The condition was first described by pioneer American pediatrician Abraham Jacobi in 1906. PVA causes areas of affected skin to appear speckled red and inflamed, yellowish and/or brown, gray or grayish-black, with scaling and a thinness that may be described as "cigarette paper". On the surface of the skin, these areas may range in size from small patches, to plaques (larger, raised areas), to neoplasms (spreading, tumor-like growths on the skin).
Large plaque parapsoriasis (also known as "parapsoriasis en plaques") are skin lesions that may be included in the modern scheme of cutaneous conditions described as parapsoriasis. These lesions, called plaques, may be irregularly round-shaped to oval and are or larger in diameter. They can be very thin plaques that are symptomatic or mildly pruritic. Large-plaque parapsoriasis is a common associate of retiform parapsoriasis, can be accompanied by poikiloderma vasculare atrophicans, and can in rare occasions be a precursor to cutaneous T-cell lymphoma.
PVA fits within this updated view of parapsoriasis as a syndrome often associated with large plaque parapsoriasis and, or including its variant form, retiform parapsoriasis. Additionally, it may be considered a precursor or variant of the lymphomatous skin disorder mycosis fungoides, which is also associated with large plaque parapsoriasis. Large plaque parapsoriasis consists of inflamed, oddly discolored (such as yellow or blue), web-patterned and scaling plaques on the skin, or larger in diameter. When the condition of the skin encompassed by these plaques worsens and becomes atrophic, it is typically considered retiform parapsoriasis. PVA can occur in either the large plaque or retiform stage, but it can only be considered PVA when its three constituents (poikiloderma, telangiectasia, atrophy) are present. PVA is therefore considered an independent syndrome identified by its constituents, wherever it occurs.